Effects of inhibiting SLC31A1 expression on alcoholic fatty liver disease in mice by regulating copper homeostasis and its underlying mechanisms
Medical Journal of Chinese People′s Liberation ArmyVol. 51, Issue 4, Pages: 567-575(2026)
Basic Research|更新时间:2026-05-15
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Effects of inhibiting SLC31A1 expression on alcoholic fatty liver disease in mice by regulating copper homeostasis and its underlying mechanisms
作者机构:
1.河南科技大学基础医学与法医学院,河南洛阳 471000
2.河南省肝病防治工程技术研究中心,河南洛阳 471000
3.河南科技大学临床医学院,河南洛阳 471000
作者简介:
E-mail: sanqiangli2001@163.com
基金信息:
the Key Scientific Research Project of Henan Colleges and Universities(23ZX006);the Henan Provincial Central Leading Local Science and Technology Development Fund Project(Z20231811030)
Sun Yue,Li San-Qiang,Zhao Ya-Di,et al.Effects of inhibiting SLC31A1 expression on alcoholic fatty liver disease in mice by regulating copper homeostasis and its underlying mechanisms[J].Medical Journal of Chinese People′s Liberation Army,2026,51(04):567-575.
Sun Yue,Li San-Qiang,Zhao Ya-Di,et al.Effects of inhibiting SLC31A1 expression on alcoholic fatty liver disease in mice by regulating copper homeostasis and its underlying mechanisms[J].Medical Journal of Chinese People′s Liberation Army,2026,51(04):567-575.DOI: 10.11855/j.issn.0577-7402.1623.2026.0228.
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